Can i get nitrous oxide

Side effects of N2O include transient dizziness, dissociation, disorientation, loss of balance, impaired memory and cognition, and weakness in the legs. When intoxicated accidents like tripping and falling may occur. Some fatal accidents have been reported due to due to asphyxia hypoxia. Heavy or sustained use of N2O inactivates vitamin B12, resulting in a functional vitamin B12 deficiency and initially causing numbness in fingers, which may further progress to peripheral neuropathy and megaloblastic anemia.

N2O use does not seem to result in dependence. Anecdotal evidence suggests that combining nitrous oxide with other drugs such as cannabis , ketamine , LSD , magic mushroom and salvia can cause intense dissociation. When inhaling directly from tanks or whippets bulbs , the gas is intensely cold C degrees and can cause frostbite to the nose, lips and throat including vocal cords. Releasing the nitrous oxide into a balloon helps to warm the gas and normalise the pressure before inhaling.

People can also harm themselves if they use faulty gas dispensers, which may explode. There are no significant withdrawal symptoms apart from cravings to use more nitrous.

If your use of nitrous oxide is affecting your health, family, relationships, work, school, financial or other life situations, you can find help and support. Not sure what you are looking for? Try our intuitive Path2Help tool and be matched with support information and services tailored to you. N2O , NOS , balloons , bulbs , buzz bomb , hippy crack , laughing gas , nangs , nitro , whippet. Nitrous oxide. Last published: November 10, What is nitrous oxide? Other types of dissociatives Ketamine Methoxetamine.

Effects of nitrous oxide There is no safe level of drug use. If a large amount of nitrous oxide is inhaled it can produce: 2,4,7 loss of blood pressure fainting heart attack.

He had no pain, no sphincter dysfunction and no autonomic symptoms. On examination, his cranial nerve function was normal. He walked with a broad-based gait, and Romberg's sign was positive. His muscle tone was flaccid, and he had mild, symmetrical, distal upper limb weakness finger abduction, thumb abduction with symmetrical mild proximal hip flexion and marked distal lower limb weakness.

Deep tendon reflexes were absent and plantar responses flexor. Sensation to light touch and pinprick was reduced below the knees, vibration sense was absent below the anterior superior iliac spines and joint position sense absent below the ankles. Initial investigations included normal full blood count, renal, liver, thyroid and bone profiles. Cerebrospinal fluid CSF was acellular with protein 0. Nerve conduction studies showed slowed motor conduction velocities and delayed or absent F-waves, consistent with a demyelinating neuropathy.

Sensory conduction was normal. MR scan of the whole spine was normal. He started treatment with intravenous immunoglobulin 0.

We started treatment with intramuscular vitamin B CASE 2 : A year-old man was admitted to the emergency department with a 6-week progressive history of bilateral lower limb weakness. He had a history of recurrent patellar dislocations for which he attended the emergency department regularly. He also had a history of depression and self-harm, with cutting behaviours and parasuicide. He took no medication, drank no alcohol and did not smoke.

On examination, his muscle tone was flaccid and he had symmetrical proximal and distal weakness of the lower and upper limbs with a glove and stocking sensory loss. Initial blood tests were normal, including full blood count, serum B 12 , folate and methylmalonic acid. His general practitioner had given a single dose of intramuscular vitamin B 12 the day before admission. CSF examination was normal. He was treated with intravenous immunoglobulin 0.

His nerve biopsy showed no primary demyelination but there was large myelinated fibre loss hence slow conduction velocities with multiple small vessel occlusions and ischaemic pathology without vessel infiltration. MR scan of brain and spinal cord showed hazy extensive white matter change in both cerebral hemispheres. On this occasion, he admitted to dislocating his patella repeatedly and deliberately with a fist or a hammer in order to attend hospital and to receive nitrous oxide analgesia, which he had returned to doing following his first discharge.

CASE 3 : A year-old woman presented to the emergency department having woken with numbness on the lateral aspect of her knees and legs. This progressed down her feet and up her thighs and abdomen over two weeks, changing in character to a burning discomfort. MR scan of brain and whole spine and nerve conduction studies were normal. CSF was acellular with protein and glucose within normal limits.

Over the next month, her symptoms progressed to involve her right hand; she developed Lhermitte's symptom, urinary urgency and required a stick to walk. On repeat examination, there was proximal upper and lower limb weakness, brisk tendon reflexes with pathological reflexes but absent ankle jerks and mute plantar responses.

Her gait was ataxic with positive Romberg's sign.